Read e-book online Clinical neurophysiology of sleep disorders PDF

By Guilleminault C. (ed.)

ISBN-10: 0444515178

ISBN-13: 9780444515179

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1993). The increased sympathetic tone during REM sleep results in vasoconstriction and subsequent reduced muscle blood flow in skeletal muscle vessels. , 1997). , 1991). 9. Endocrine physiology The secretory activity of the endocrine system displays a specific regulation during NREM sleep and REM sleep. 2. Cutaneous circulation The onset of NREM sleep results in a drop in the set point temperature which results in a vasodilatation in skin vasculature. During REM sleep, a cold environment results in an increase in skin blood flow due to a decrease in neurogenic vasoconstriction.

J. Clin. Endocrinol. , 80(1): 214–219. Spiegel, K, Follenius, M, Simon, C, et al. (1994) Prolactin secretion and sleep. Sleep, 17(1): 20–27. Spiegel, K, Luthringer, R, Follenius, M, et al. (1995) Temporal relationship between prolactin secretion and NREM–REM SLEEP slow-wave electroencephalic activity during sleep. Sleep, 18(7): 543–548. Steriade, M (1994) Sleep oscillations and their blockage by activating systems. J. , 19(5): 354–358. Steriade, M (2003) The corticothalamic system in sleep. Front.

The ROC channel (ROC/A1) and the second EEG (O2/A1) channels are contaminated with ECG artifact. The artifact can be identified by aligning the spike-like activity noted in channels with the R wave on the ECG channel. It is localized to the A1 electrode because it is seen in both ROC/A1 and O2/A1 channels and A1 is common to both channels. It should be noted that the high-amplitude ECG artifact, seen in the EMG channel below the ECG channel, is unavoidable. This artifact is due to the proximity of EMG electrodes to the heart, which creates a robust signal superimposed on the intercostal EMG signal.

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Clinical neurophysiology of sleep disorders by Guilleminault C. (ed.)


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